Necroinflammation |
necroptosome |
MIP-1-alpha receptor-like-1 |
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This term (also: programmed necrosis, regulated necrosis) has been used to distinguish stimulus-dependent necrosis that proceeds in the absence of caspase activation from those necrotic events induced by nonspecific traumas (e.g., heat shock) or secondary necrosis induced as a consequence of cell death by apoptosis. In several experimental models, programmed necrosis/necroptosis has been shown to be a crucial control point for pathogen- or injury-induced inflammation (Challa and Chan, 2010). Other terms, such as aponecrosis or necrapoptosis probably mean the same thing, with all terms referring to the observation that
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